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361 However, in a few apical patches the channel density was increased X 3, which could indicate channel clustering. Ca fluorescence imaging shows that application of T-channel antagonists reduces the Ca influx associated with backpropagating action potentials, and has a two-fold greater effect in the dendrites than in the soma
362 However, in a few apical patches the channel density was increased X 3, which could indicate channel clustering. Ca fluorescence imaging shows that application of T-channel antagonists reduces the Ca influx associated with backpropagating action potentials, and has a two-fold greater effect in the soma than in the dendrites
363 However, in guinea pigs, a combination of high-speed imaging and simultaneous intracellular recordings showed that direct depolarization of the soma or dendrites never caused dendritic [Na+]i increases, suggesting that the climbing fiber-activated [Na+]i changes in the dendrites are due to Na+ entry through ligand-gated channels
364 However, recordings in slices showed that D1 receptor activation can either inhibit or enhance evoked activity, depending on the level of membrane depolarization, by modulating an L-type Ca2+ conductance
365 I M may contribute to a sag and rebound of voltage response to hyperpolarizing current steps
366 Ia EPSPs are mediated largely by AMPA receptors (muscle afferents:
367 Ia IPSPs are chloride mediated
368 Ia IPSPs are readily affected by soma current or Cl- injection, indicating location at the soma or proximal dendrites
369 Ia synapses are immunoreactive for GLU
370 Identification of subunit mRNAs
371 Ih conductance causes voltage attunuation and is more concentrated in dendrites than in soma
372 Ih current: slowly developing hyperpolarisation-activated current with a threshold generally positive to resting potential and with a strongly voltage-dependent activation time constant. The current was Na+- and K+-sensitive, suppressed by external Cs+, and insensitive to Ba++. The Ih should be tonically active at rest, and may contribute to the oscillatory behaviour of the bulbar network
373 Immunochemical evidence of glutamate as neurotransmitter in the terminals of parallel fibers
374 Immunocytochemistry and in situ hybridisation showed that NMDAR1 was expressed in most CN neuronal types, including the fusiform cell apical dendrites. However, fusiform cell basal dendrites, which are the synaptic sites of cochlear nerve fibers, did not express NMDAR1
375 Immunohistochemical evidence shows that CaV1.3 (an L type channel; seeMembrane Properties Resource) is present in soma and all dendritic compartments in turtle motoneurons
376 Immunohistochemical evidence shows that CaV1.3 is present in soma and all dendritic compartments in turtle motoneurons
377 Immunolabeling was observed in soma and dendrites of layer V pyramidal cells in the frontal cortex
378 Implied by current clamp recording of action potential at soma
379 Implied by data on more proximal dendritic regions; still to be tested
380 Implied by Glu released by other compartments of the mitral cell (Dale's law). Target (destination) is presumably PG cell dendrites in the glomerulus
381 Implied by recording of fast prepotential. Dual patch recordings provide evidence for both backpropagating and forward-propagating impulses in the primary dendrite
382 Implied from evidence that local interneurons colocalize FMRFamide and GABA
383 Importance for membrane excitability of an GABAB receptor-activated inward-rectifying potassium current, sensitive to pertussis toxin and barium
384 In a study of acutely isolated rat cells under whole cell recording across development states (Day 6 - Day 29), it was found that delayed rectifier currents decayed along a double-exponential time course and were 50% blocked by TEA (tetraethylammonium, a I(K) antagonist) at +30 mV at a concentration of about 1mM, as well as being partially blocked by 4-AP (4-aminopyridine). The current also appeared to increase over this development period. This increase was approximately 300% much larger in CA1 cells than in CA3 cells, with only approximately 50%
385 In a study of acutely isolated rat cells under whole cell recording across development states (Day 6 - Day 29), it was found that delayed rectifier currents decayed along a double-exponential time course and were 50% blocked by TEA (tetraethylammonium, a K(DR) antagonist) at +30 mV at a concentration of about 1mM, as well as being partially blocked by 4-AP (4-aminopyridine). The current also appeared to increase over this development period. This increase was approximately 300% much larger in CA1 cells than in CA3 cells, with only approximately 50%
386 In a study of acutely isolated rat cells under whole cell recording across development states (Day 6 - Day 29), it was found that delayed rectifier currents decayed along a double-exponential time course and were 50% blocked by TEA (tetraethylammonium, a K(DR) antagonist) at +30 mV at a concentration of about 1mM, as well as being partially blocked by 4-AP (4-aminopyridine). The current also appeared to increase over this development period. This increase was approximately 300% much larger in CA1 cells than in CA3 cells, with only approximately 50%
387 In a study of acutely isolated rat cells under whole cell recording across developmental states (Day 6 - Day 29), I(A) was separated from I(K) by subtraction methods. It was found that I(A) was rapidly activated and inactivated and was 80% blocked by 4-AP (4-aminopyridine), but was insensitive to TEA (tetraethylammonium) and dendrotoxin (Klee et al, 1995). The current has also been shown to be modulated by K concentration (Eder et al, 1996), though this effect appears to be restricted to very early in development (Klee et al, 1997).
388 In addition, IP3 gated ion channel; found in the autodendritic membrane
389 In an immunocytochemical study in zebrafish 60-70% of cells showed KA receptor mediated labelling
390 In an immunocytochemical study in zebrafish 60-70% of cells showed KA receptor mediated labelling
391 In an immunocytochemical study in zebrafish all cells resulted in NMDA receptor mediated labelling
392 In cell-attached patch-clamp recordings from the soma in guinea pig hippocampal slices, L-currents were found in 34% of the patches and found to have single channel conductances of 23-27 pS
393 In cell-attached patch-clamp recordings from the soma in guinea pig hippocampal slices, L-currents were found in 34% of the patches and found to have single channel conductances of 23-27 pS (Johnston et al. 1992;
394 In cell-attached patch-clamp recordings from the soma in guinea pig hippocampal slices, N-currents were found in 63% of the patches and found to have single channel conductances of 14 pS (Johnston et al. 1992;
395 In cell-attached patch-clamp recordings from the soma in guinea pig hippocampal slices, T-currents were found in 72% of the patches and found to have single channel conductances of 8 pS
396 In cell-attached patch-clamp recordings from the soma in guinea pig hippocampal slices, T-currents were found in 72% of the patches and found to have single channel conductances of 8 pS (Johnston et al. 1992,
397 In cilia; K 0.5 for Ca activation is 5 uM; most of Ca activated current is carried by chloride and persists in the absence of Na and K; Cl channel inhibitor 3',5-dichlorodiphenylamine-2-carboxylate (300uM) reduces current 90%; other Cl-channel inhibitors were tested [SITS, DIDS, A9C, DPC, NPPB, DCDPC]
398 In contrast, a study using radioactive in situ hybridization histochemistry looked at mRNA coding an NMDA glutamate binding protein and at NMDAR1 (an NMDAR subunit) expression and found heavy labeling for both in the pyramidal and polymorphic layers but little in the molecular layer
399 In gold fish retinal bipolar cells, four currents are observed: Ca currents, voltage- and calcium-dependent K currents, and Ih current
400 In mouse retinal bipolar cells, T-type Ca currents were recorded in soma, while L-type currents were recorded from axonal terminal