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201 by contrast,
202 By using dendritic recordings and calcium transients in rats it was shown that this current may control AP propagation in lateral dendrites
203 Ca -dependent Chloride current at the presynaptic terminals of goldfish retinal bipolar cells
204 Ca fluorescence imaging shows that application of L-channel antagonists reduces the Ca influx associated with backpropagating action potentials, and has a significantly greater effect in the proximal dendrites than in more distal dendrites
205 Ca fluorescence imaging shows that application of N-channel antagonists slightly reduces the Ca influx associated with backpropagating action potentials
206 Ca fluorescence imaging shows that application of P-channel antagonists reduces the Ca influx associated with backpropagating action potentials
207 Ca-dependent potassium currents are seen in dissociated cells
208 CA1 neurons and subiculum neurons in hippoampus differ in firing pattern (the former being regular and the later being either regular, weakly bursting or strongly bursting) and resting membrane properties (such as input restistance and membran time constant); however, low concentration of 4-AP (50 &#181M) can convert neurons in both regions into firing bursting action potentials
209 CA1 neurons and subiculum neurons in hippoampus differ in firing pattern (the former being regular and the later being either regular, weakly bursting or strongly bursting) and resting membrane properties (such as input restistance and membran time constant); however, low concentration of 4-AP (50 ?M) can convert neurons in both regions into firing bursting action potentials
210 CA1 neurons and subiculum neurons in hippoampus differ in firing pattern (the former being regular and the later being either regular, weakly bursting or strongly bursting) and resting membrane properties (such as input restistance and membran time constant); however, low concentration of 4-AP (50 µM) can convert neurons in both regions into firing bursting action potentials
211 CA1 pyramidal neurons increase their firing (recorded extracellularly) in response to ionophoresed Glu within their apical dendritic fields or in the cell body layer (Dudar 1974 PMID#4437726).
212 Ca2+ -activated K+ current at presynaptic terminals of goldfish retinal bipolar cells
213 Ca2+-activated K+ currents were studied using whole-cell patch-clamp recordings from freshly dissociated mouse neocortical pyramidal neurons
214 Calcium entry through N-type calcium channels (CaV2.2) causes activation of KCa channels that decrease the firing rate and increase the regularity of firing in SNr GABA neurons”
215 Calcium influx through NMDA receptors can directly trigger presynatic GABA release for local dendrodendritic feedback inhibition. DDI is elicited by photorelease of caged Ca++, with and without Cd++ and Ni++.
216 Calcium influx through NMDA receptors directly evokes GABA release in granule cells.
217 Calcium is involved in delayed release of neurotransmitter at synapses between granule cell their postsynaptic targets (stellate cells and Purkinje cells)
218 Calcium-dependent potassium channels are preferentially activated by calcium entry through N- and Q-type channels
219 CaV channels are among the intrinsic membrane channels influencing the excitability of the SNr neuronal membrane. CaV channels have been shown to influence spontaneous activity in these neurons, as well as more complex behaviors such as plateau potential generation burst firing.”
220 Cell-attached dendritic recordings in rats up to about 60um from the soma showed that low-threshold calcium channels were concentrated at proximal dendritic locations, sites known to receive excitatory synaptic connections from primary afferents, suggesting that they play a key role in the amplification of sensory inputs to TC neurons
221 Cell-attached patches on the proximal 100um of the apical dendrite did not contain sAHP channels. Amputation of the apical dendrite approximately 30 micron from the soma, while simultaneously recording the sAHP whole cell current at the soma, depressed the sAHP amplitude by only approximately 30% compared with control. Somatic cell-attached and nucleated patches did not contain sAHP current. Amputation of the axon about 20um from the soma had little effect on the amplitude of the sAHP. By this process of elimination, it is suggested that sAHP channels may be concentrated in the basal dendrites of CA1 pyramids
222 Cell-attached recordings in rats up to about 60um from the soma demonstrated a non-uniform dendritic distribution of channels
223 Cell-attached recordings in rats up to about 60um from the soma demonstrated a roughly uniform density of channels across the somatodendritic area examined that corresponds to approximately half the average path length of TC neuron dendrites
224 Cell-specifc modulation of GABAA receptor- mediated chloride current by dopamine. In interneurons (mainly granule cells), dopamine reduces GABAA Cl- current, via D1 receptor and involves phosphorylation of GABAA receptors by PKA. In mitral cell, dopamine enhances GABA responses via activation of D2 receptors and phosphorylation of GABAA receptors via PKC.
225 Cell-specifc modulation of GABAA receptor- mediated chloride current by dopamine. In interneurons (mainly granule cells), dopamine reduces GABAA Cl- current, via D1 receptor and involves phosphorylation of GABAA receptors by PKA. In mitral cell, dopamine enhances GABA responses via activation of D2 receptors and phosphorylation of GABAA receptors via PKC.
226 Cells acutely dissociated from slices obtained from chronic temporal lobe epilepsy patients displayed a high-voltage activated Ca2+ conductance with a pronounced Ca2+-dependent inactivation
227 Cells were voltage-clamped using a single microelectrode, at 23-30 degrees C. M-current resembled that of sympathetic ganglion cells. It was abolished by addition of carbachol, muscarine or bethanechol, as well as by 1 mM barium. It was suggested that activation of cholinergic septal inputs to the hippocampus facilitates repetitive firing of pyramidal cells by turning off the M-conductance, without much change in the resting potential of the cell
228 Cerebellar granule cells from young rats (postnatal days 1-9) possess voltage-activated inward Na+ current as well as two types of K+ current, IA and IK
229 Chen et al 1997). Dendritic patch recordings showed an even density of Na channels (120pS um-2) up to 350 um from the soma along the primary dendrite to the origin of the glomerular tuft
230 Chen et al 1997).Dendritic patch recordings showed an even density of Na channels (120pSum-2) up to 350 um from the soma along the primary dendrite to theorigin of the glomerular tuft
231 Choline acetyltransferase immunocytochemistry shows that cholinergic projections from the basal forebrain to the main olfactory bulb focus synaptic innervation on interneurons, on the dendritic spines of periglomerular and granule cells.
232 Cholinergic agonist carbachol caused a significant suppression of inhibitory postsynaptic potentials (IPSPs) in the pyramidal neurons that were induced by stimulation of layer Ib, with a weaker effect on IPSPs induced by stimulation of layer Ia
233 Cholinergic agonist carbachol selectively suppresses intrinsic fiber synaptic potentials but not afferent fiber synaptic potentials.
234 Cholinergic agonist carbachol selectively suppresses intrinsic fiber synaptic potentials but not afferent fiber synaptic potentials. A presynaptic mechanism of cholinergic suppression is suggested
235 Cholinergic agonist carbachol selectively suppresses intrinsic fiber synaptic potentials but not afferent fiber synaptic potentials. (Hasselmo ME and Bower JM, 1992353 ) (Patil MM and Hasselmo ME, 1999341 ).
236 Cholinergic interneurones in rat exhibit a ATP-sensitive potassium channel current. sigle-cell-RT-PCR shows that this channel is formed from Kir6.1 and SUR1 subunits
237 cited in Johnston and Amaral, 1998).
238 Clusters of glycine receptors were found on the somatodendritic membranes of Alpha ganglion cells
239 Clusters of the alpha1, and alpha2, alpha3, and gamma2 subunits of the GABAA receptor were found on the somatodendritic membranes of Alpha ganglion cells. Experiments with different combinations of the subunit-specific antibodies showed that the alpha1, alpha2, and alpha3 subunits of the GABA(A) receptor are not colocalized within the same clusters, suggesting that an individual neuron can express several isoforms of the GABAA receptor and that these different isoforms are aggregated at distinct postsynaptic sites
240 Combining preembedding and postembedding immunostaining at the EM level, GluR2/3 and NMDAR1 immunoreactivity was located in somata and in proximal and distal dendrites