Name
1	A beta 25-35-induced depression of long-term potentiation in area CA1 in vivo and in vitro is attenuated by verapamil.
2	Abeta(25-35) and Abeta(1-40) act on different calcium channels in CA1 hippocampal neurons.
3	Activation of the L voltage-sensitive calcium channel by mitogen-activated protein (MAP) kinase following exposure of neuronal cells to beta-amyloid. MAP kinase mediates beta-amyloid-induced neurodegeneration.
4	Alterations in synaptic transmission and long-term potentiation in hippocampal slices from young and aged PDAPP mice.
5	AMPA receptor downscaling at the onset of Alzheimer's disease pathology in double knockin mice.
6	Amyloid beta peptide as a physiological modulator of neuronal 'A'-type K(+) current.
7	Amyloid beta protein potentiates Ca2+ influx through L-type voltage-sensitive Ca2+ channels: a possible involvement of free radicals.
8	beta-Amyloid increases dendritic Ca2+ influx by inhibiting the A-type K+ current in hippocampal CA1 pyramidal neurons.
9	Blockade of long-term potentiation by beta-amyloid peptides in the CA1 region of the rat hippocampus in vivo.
10	Cortical Synaptic Integration In Vivo Is Disrupted by Amyloid-{beta} Plaques
11	Dendritic changes in Alzheimer's disease and factors that may underlie these changes
12	Diffusible, nonfibrillar ligands derived from Abeta1-42 are potent central nervous system neurotoxins.
13	Early phenotypic changes in transgenic mice that overexpress different mutants of amyloid precursor protein in brain.
14	Effect of beta-amyloid block of the fast-inactivating K+ channel on intracellular Ca2+ and excitability in a modeled neuron.
15	Effect of stimulation of the horizontal limb of the diagonal band on rat olfactory bulb neuronal activity.
16	Frequency potentiation of a central cholinergic circuit.
17	Huperzine A inhibits the sustained potassium current in rat dissociated hippocampal neurons.
18	Huperzine A, a nootropic agent, inhibits fast transient potassium current in rat dissociated hippocampal neurons.
19	Impaired synaptic plasticity and learning in aged amyloid precursor protein transgenic mice.
20	In vivo synaptic transmission in young and aged amyloid precursor protein transgenic mice.
21	Individuals with Down's syndrome demonstrate abnormal olfactory event-related potentials.
22	Inhibition of L-type voltage dependent calcium channels causes impairment of long-term potentiation in the hippocampal CA1 region in vivo.
23	M1 receptors play a central role in modulating AD-like pathology in transgenic mice
24	Mechanism of impairment of long-term potentiation by amyloid beta is independent of NMDA receptors or voltage-dependent calcium channels in hippocampal CA1 pyramidal neurons.
25	Neuronal deficiency of presenilin 1 inhibits amyloid plaque formation and corrects hippocampal long-term potentiation but not a cognitive defect of amyloid precursor protein [V717I] transgenic mice.
26	Nicotine enhances the depressive actions of A beta 1-40 on long-term potentiation in the rat hippocampal CA1 region in vivo.
27	Okadaic acid mediates tau phosphorylation via sustained activation of the L-voltage-sensitive calcium channel.
28	Olfactory bulb output neurons excited from a basal forebrain magnocellular nucleus.
29	Olfactory event-related potentials in Alzheimer's disease.
30	p35/Cdk5 pathway mediates soluble amyloid-beta peptide-induced tau phosphorylation in vitro.
31	Plaque-independent disruption of neural circuits in Alzheimer's disease mouse models.
32	Presenilin 1 deficiency alters the activity of voltage-gated Ca2+ channels in cultured cortical neurons
33	Protofibrillar intermediates of amyloid beta-protein induce acute electrophysiological changes and progressive neurotoxicity in cortical neurons.
34	Selective modulation of GABAA receptors by aluminum.
35	Similar levels of long-term potentiation in amyloid precursor protein -null and wild-type mice in the CA1 region of picrotoxin treated slices.
36	Soluble oligomers of beta amyloid (1-42) inhibit long-term potentiation but not long-term depression in rat dentate gyrus.
37	Synaptic plasticity in animal models of early Alzheimer's disease.
38	The channel hypothesis of Alzheimer's disease: current status.