BrainPharm: Computational model - Breakdown of accmmodation in nerve: a possible role for INAp (Hennings et al 2005)

Computational model

Data

Name

Breakdown of accmmodation in nerve: a possible role for INAp (Hennings et al 2005)

Submitter name

Pipeline ID

Model File

BOA Model [?]

Notes

The present modeling study suggests that persistent, low-threshold, rapidly activating sodium currents have a key role in breakdown of accommodation, and that breakdown of accommodation can be used as a tool for studying persistent sodium current under normal and pathological conditions. See paper for more and details.

Model Neurons

Spinal cord lumbar motor neuron alpha ACh cell Show Other

More Cells

Myelinated neuron Show Other

Model Currents

I Na,p Show Other
I Na,t Show Other
I K Show Other

Model Receptors

Model Type

Neuron or other electrically excitable cell Show Other

Model Concept

Action Potential Initiation Show Other
Action Potentials Show Other
Pathophysiology Show Other
Electrotonus Show Other

Simulator software

MATLAB Show Other

Model papers

Hennings K, Arendt-Nielsen L, Andersen OK (2005) Show Other

Gene

hg folder name (applicable when in ModelDB hg)

Region Organism

Model ID Number

Gap Junctions

Implemented by

Hennings, Kristian [krist at hst.auc.dk] Show Other

Public Submitter Email Address

Other Neuron

Model Neurotransmitters

Other Receptor

Other Current

Other Neurotransmitter

Other Model Type

Other Concept

Other Simulator

Other Implementer

Alternative Version

Citation

Default File Notes

Other Gene

Simulation Platform ID

Has Model View

False

Component

RAM

Hide Auto-launch button

False

Run Protocols

ICG Channel Details

Species

Other categories referring to Breakdown of accmmodation in nerve: a possible role for INAp (Hennings et al 2005)

Revisions:	8
Last Time:	4/13/2007 6:11:53 PM
Reviewer:	Tom Morse - MoldelDB admin
Owner:	Tom Morse - MoldelDB admin