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Thalamic quiescence of spike and wave seizures (Lytton et al 1997)
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Accession:
9889
A phase plane analysis of a two cell interaction between a thalamocortical neuron (TC) and a thalamic reticularis neuron (RE).
Reference:
1 .
Lytton WW, Contreras D, Destexhe A, Steriade M (1997) Dynamic interactions determine partial thalamic quiescence in a computer network model of spike-and-wave seizures.
J Neurophysiol
77
:1679-96
[
PubMed
]
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Model Information
(Click on a link to find other models with that property)
Model Type:
Realistic Network;
Brain Region(s)/Organism:
Thalamus;
Cell Type(s):
Thalamus geniculate nucleus/lateral principal GLU cell;
Thalamus reticular nucleus GABA cell;
Channel(s):
I T low threshold;
Gap Junctions:
Receptor(s):
GabaA;
Glutamate;
Gene(s):
Transmitter(s):
Gaba;
Glutamate;
Simulation Environment:
NEURON;
Model Concept(s):
Temporal Pattern Generation;
Oscillations;
Calcium dynamics;
Implementer(s):
Lytton, William [bill.lytton at downstate.edu];
Destexhe, Alain [Destexhe at iaf.cnrs-gif.fr];
Search NeuronDB
for information about:
Thalamus geniculate nucleus/lateral principal GLU cell
;
Thalamus reticular nucleus GABA cell
;
GabaA
;
Glutamate
;
I T low threshold
;
Gaba
;
Glutamate
;
/
lytton97
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