Citation Relationships

Legends: Link to a Model Reference cited by multiple papers


Crunelli V, Leresche N (2002) Childhood absence epilepsy: genes, channels, neurons and networks. Nat Rev Neurosci 3:371-82 [PubMed]

References and models cited by this paper

References and models that cite this paper

Broicher T, Kanyshkova T, Landgraf P, Rankovic V, Meuth P, Meuth SG, Pape HC, Budde T (2007) Specific expression of low-voltage-activated calcium channel isoforms and splice variants in thalamic local circuit interneurons. Mol Cell Neurosci 36:132-45 [Journal] [PubMed]
Broicher T, Kanyshkova T, Meuth P, Pape HC, Budde T (2008) Correlation of T-channel coding gene expression, IT, and the low threshold Ca2+ spike in the thalamus of a rat model of absence epilepsy. Mol Cell Neurosci 39:384-99 [Journal] [PubMed]
Broicher T, Seidenbecher T, Meuth P, Munsch T, Meuth SG, Kanyshkova T, Pape HC, Budde T (2007) T-current related effects of antiepileptic drugs and a Ca2+ channel antagonist on thalamic relay and local circuit interneurons in a rat model of absence epilepsy. Neuropharmacology 53:431-46 [Journal] [PubMed]
Chen M, Guo D, Li M, Ma T, Wu S, Ma J, Cui Y, Xia Y, Xu P, Yao D (2015) Critical Roles of the Direct GABAergic Pallido-cortical Pathway in Controlling Absence Seizures. PLoS Comput Biol 11:e1004539 [Journal] [PubMed]
   Basal ganglia-corticothalamic (BGCT) network (Chen et al., 2014) [Model]
Chen M, Guo D, Wang T, Jing W, Xia Y, Xu P, Luo C, Valdes-Sosa PA, Yao D (2014) Bidirectional control of absence seizures by the basal ganglia: a computational evidence. PLoS Comput Biol 10:e1003495 [Journal] [PubMed]
   Basal ganglia-corticothalamic (BGCT) network (Chen et al., 2014) [Model]
Hall SP, Traub RD, Adams NE, Cunningham MO, Schofield I, Jenkins AJ, Whittington MA (2018) Enhanced interlaminar excitation or reduced superficial layer inhibition in neocortex generates different spike-and-wave-like electrographic events in vitro. J Neurophysiol 119:49-61 [Journal] [PubMed]
   The origin of different spike and wave-like events (Hall et al 2017) [Model]
Hayut I, Fanselow EE, Connors BW, Golomb D (2011) LTS and FS inhibitory interneurons, short-term synaptic plasticity, and cortical circuit dynamics. PLoS Comput Biol 7:e1002248 [Journal] [PubMed]
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Liu Y, Milton J, Campbell SA (2019) Outgrowing seizures in Childhood Absence Epilepsy: time delays and bistability. J Comput Neurosci [Journal] [PubMed]
   Thalamocortical loop with delay for investigation of absence epilepsy (Liu et al 2019) [Model]
Suffczynski P, Kalitzin S, Lopes Da Silva FH (2004) Dynamics of non-convulsive epileptic phenomena modeled by a bistable neuronal network. Neuroscience 126:467-84 [Journal] [PubMed]
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Vitko I, Chen Y, Arias JM, Shen Y, Wu XR, Perez-Reyes E (2005) Functional characterization and neuronal modeling of the effects of childhood absence epilepsy variants of CACNA1H, a T-type calcium channel. J Neurosci 25:4844-55 [Journal] [PubMed]
   T channel currents (Vitko et al 2005) [Model]
Zhao X, Robinson PA (2015) Generalized seizures in a neural field model with bursting dynamics. J Comput Neurosci 39:197-216 [Journal] [PubMed]
   Neural-field model of generalized seizures (Zhao et al., 2015) [Model]
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