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Wittenberg GM, Wang SS (2006) Malleability of spike-timing-dependent plasticity at the CA3-CA1 synapse. J Neurosci 26:6610-7 [PubMed]

References and models cited by this paper

References and models that cite this paper

Graupner M, Brunel N (2007) STDP in a bistable synapse model based on CaMKII and associated signaling pathways. PLoS Comput Biol 3:e221 [Journal] [PubMed]
   CaMKII system exhibiting bistability with respect to calcium (Graupner and Brunel 2007) [Model]
Graupner M, Brunel N (2012) Calcium-based plasticity model explains sensitivity of synaptic changes to spike pattern, rate, and dendritic location. Proc Natl Acad Sci U S A 109:3991-6 [Journal] [PubMed]
Kim Y, Hsu CL, Cembrowski MS, Mensh BD, Spruston N (2015) Dendritic sodium spikes are required for long-term potentiation at distal synapses on hippocampal pyramidal neurons. Elife [Journal] [PubMed]
   CA1 pyramidal neuron: Dendritic Na+ spikes are required for LTP at distal synapses (Kim et al 2015) [Model]
Saudargiene A, Cobb S, Graham BP (2015) A computational study on plasticity during theta cycles at Schaffer collateral synapses on CA1 pyramidal cells in the hippocampus. Hippocampus 25:208-18 [Journal] [PubMed]
   CA1 pyramidal neuron: synaptic plasticity during theta cycles (Saudargiene et al. 2015) [Model]
Urakubo H, Honda M, Froemke RC, Kuroda S (2008) Requirement of an allosteric kinetics of NMDA receptors for spike timing-dependent plasticity. J Neurosci 28:3310-23 [Journal] [PubMed]
   An allosteric kinetics of NMDARs in STDP (Urakubo et al. 2008) [Model]
Yu X, Shouval HZ, Knierim JJ (2008) A biophysical model of synaptic plasticity and metaplasticity can account for the dynamics of the backward shift of hippocampal place fields. J Neurophysiol 100:983-92 [Journal] [PubMed]
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