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Zhou D, Lambert S, Malen PL, Carpenter S, Boland LM, Bennett V (1998) AnkyrinG is required for clustering of voltage-gated Na channels at axon initial segments and for normal action potential firing. J Cell Biol 143:1295-304 [PubMed]

References and models cited by this paper

References and models that cite this paper

Clark BA, Monsivais P, Branco T, London M, Häusser M (2005) The site of action potential initiation in cerebellar Purkinje neurons. Nat Neurosci 8:137-9 [Journal] [PubMed]
Kole MH, Ilschner SU, Kampa BM, Williams SR, Ruben PC, Stuart GJ (2008) Action potential generation requires a high sodium channel density in the axon initial segment. Nat Neurosci 11:178-86 [Journal] [PubMed]
   Na+ channel dependence of AP initiation in cortical pyramidal neuron (Kole et al. 2008) [Model]
Meza RC, López-Jury L, Canavier CC, Henny P (2018) Role of the Axon Initial Segment in the Control of Spontaneous Frequency of Nigral Dopaminergic Neurons In Vivo. J Neurosci 38:733-744 [Journal] [PubMed]
   Role of the AIS in the control of spontaneous frequency of dopaminergic neurons (Meza et al 2017) [Model]
Moubarak E, Engel D, Dufour MA, Tapia M, Tell F, Goaillard JM (2019) Robustness to Axon Initial Segment Variation Is Explained by Somatodendritic Excitability in Rat Substantia Nigra Dopaminergic Neurons. J Neurosci 39:5044-5063 [Journal] [PubMed]
   Dopaminergic subtantia nigra neuron (Moubarak et al 2019) [Model]
Royeck M, Horstmann MT, Remy S, Reitze M, Yaari Y, Beck H (2008) Role of axonal NaV1.6 sodium channels in action potential initiation of CA1 pyramidal neurons. J Neurophysiol 100:2361-80 [Journal] [PubMed]
   Axonal NaV1.6 Sodium Channels in AP Initiation of CA1 Pyramidal Neurons (Royeck et al. 2008) [Model]
Vascak M, Sun J, Baer M, Jacobs KM, Povlishock JT (2017) Mild Traumatic Brain Injury Evokes Pyramidal Neuron Axon Initial Segment Plasticity and Diffuse Presynaptic Inhibitory Terminal Loss. Front Cell Neurosci 11:157 [Journal] [PubMed]
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