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Staley KJ, Proctor WR (1999) Modulation of mammalian dendritic GABA(A) receptor function by the kinetics of Cl- and HCO3- transport. J Physiol 519 Pt 3:693-712 [PubMed]

References and models cited by this paper

References and models that cite this paper

Buchin A, Chizhov A, Huberfeld G, Miles R, Gutkin BS (2016) Reduced Efficacy of the KCC2 Cotransporter Promotes Epileptic Oscillations in a Subiculum Network Model. J Neurosci 36:11619-11633 [Journal] [PubMed]
   Subiculum network model with dynamic chloride/potassium homeostasis (Buchin et al 2016) [Model]
Doyon N, Prescott SA, Castonguay A, Godin AG, Kröger H, De Koninck Y (2011) Efficacy of synaptic inhibition depends on multiple, dynamically interacting mechanisms implicated in chloride homeostasis. PLoS Comput Biol 7:e1002149 [Journal] [PubMed]
Jedlicka P, Deller T, Gutkin BS, Backus KH (2011) Activity-dependent intracellular chloride accumulation and diffusion controls GABA(A) receptor-mediated synaptic transmission. Hippocampus 21:885-98 [Journal] [PubMed]
   Effects of Chloride accumulation and diffusion on GABAergic transmission (Jedlicka et al 2011) [Model]
Lewin N, Aksay E, Clancy CE (2012) Computational modeling reveals dendritic origins of GABA(A)-mediated excitation in CA1 pyramidal neurons. PLoS One 7:e47250 [Journal] [PubMed]
   Paradoxical GABA-mediated excitation (Lewin et al. 2012) [Model]
Lombardi A, Jedlicka P, Luhmann HJ, Kilb W (2019) Interactions Between Membrane Resistance, GABA-A Receptor Properties, Bicarbonate Dynamics and Cl-Transport Shape Activity-Dependent Changes of Intracellular Cl- Concentration Int J of Mol Sci, in press
   Factors contribution to GDP-induced [Cl-]i transients (Lombardi et al 2019) [Model]
Prescott SA, Sejnowski TJ, De Koninck Y (2006) Reduction of anion reversal potential subverts the inhibitory control of firing rate in spinal lamina I neurons: towards a biophysical basis for neuropathic pain. Mol Pain 2:32 [Journal] [PubMed]
Ratté S, Prescott SA (2011) ClC-2 channels regulate neuronal excitability, not intracellular chloride levels. J Neurosci 31:15838-43 [Journal] [PubMed]
   ClC-2 channels regulate neuronal excitability, not intracellular Cl- levels (Ratte & Prescott 2011) [Model]
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