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Shibata R, Nakahira K, Shibasaki K, Wakazono Y, Imoto K, Ikenaka K (2000) A-type K+ current mediated by the Kv4 channel regulates the generation of action potential in developing cerebellar granule cells. J Neurosci 20:4145-55 [PubMed]

References and models cited by this paper

References and models that cite this paper

Ashhad S, Narayanan R (2013) Quantitative interactions between the A-type K+ current and inositol trisphosphate receptors regulate intraneuronal Ca2+ waves and synaptic plasticity. J Physiol 591:1645-69 [Journal] [PubMed]
  • Calcium waves and mGluR-dependent synaptic plasticity in CA1 pyr. neurons (Ashhad & Narayanan 2013) [Model]
Diwakar S, Magistretti J, Goldfarb M, Naldi G, D'Angelo E (2009) Axonal Na+ channels ensure fast spike activation and back-propagation in cerebellar granule cells. J Neurophysiol 101:519-32 [Journal] [PubMed]
  • Multicompartmental cerebellar granule cell model (Diwakar et al. 2009) [Model]
Fineberg JD, Ritter DM, Covarrubias M (2012) Modeling-independent elucidation of inactivation pathways in recombinant and native A-type Kv channels. J Gen Physiol 140:513-27 [Journal] [PubMed]
  • Neurophysiological impact of inactivation pathways in A-type K+ channels (Fineberg et al 2012) [Model]
Molineux ML, Fernandez FR, Mehaffey WH, Turner RW (2005) A-type and T-type currents interact to produce a novel spike latency-voltage relationship in cerebellar stellate cells. J Neurosci 25:10863-73 [Journal] [PubMed]
  • IA and IT interact to set first spike latency (Molineux et al 2005) [Model]
Zheng Y, Liu P, Bai L, Trimmer JS, Bean BP, Ginty DD (2019) Deep Sequencing of Somatosensory Neurons Reveals Molecular Determinants of Intrinsic Physiological Properties Neuron [Journal]
  • DRG neuron models investigate how ion channel levels regulate firing properties (Zheng et al 2019) [Model]
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