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•	Subiculum network model with dynamic chloride/potassium homeostasis (Buchin et al 2016) [Model]

Chen K, Aradi I, Thon N, Eghbal-Ahmadi M, Baram TZ, Soltesz I (2001) Persistently modified h-channels after complex febrile seizures convert the seizure-induced enhancement of inhibition to hyperexcitability. Nat Med 7:331-7 [Journal] [PubMed]

•	Febrile seizure-induced modifications to Ih (Chen et al 2001) [Model]

De Schutter E, Smolen P (1998) Calcium dynamics in large neuronal models Methods In Neuronal Modeling: From Ions To Networks, Koch C:Segev I, ed. pp.211

Doyon N, Prescott SA, Castonguay A, Godin AG, Kröger H, De Koninck Y (2011) Efficacy of synaptic inhibition depends on multiple, dynamically interacting mechanisms implicated in chloride homeostasis. PLoS Comput Biol 7:e1002149 [Journal] [PubMed]

Gentiletti D, Suffczynski P, Gnatkovsky V, de Curtis M (2017) Changes of Ionic Concentrations During Seizure Transitions - A Modeling Study. Int J Neural Syst 27:1750004 [Journal] [PubMed]

•	Changes of ionic concentrations during seizure transitions (Gentiletti et al. 2016) [Model]

Jedlicka P, Deller T, Gutkin BS, Backus KH (2011) Activity-dependent intracellular chloride accumulation and diffusion controls GABA(A) receptor-mediated synaptic transmission. Hippocampus 21:885-98 [Journal] [PubMed]

•	Effects of Chloride accumulation and diffusion on GABAergic transmission (Jedlicka et al 2011) [Model]

Kapur A, Pearce RA, Lytton WW, Haberly LB (1997) GABAA-mediated IPSCs in piriform cortex have fast and slow components with different properties and locations on pyramidal cells. J Neurophysiol 78:2531-45 [Journal] [PubMed]

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•	Paradoxical GABA-mediated excitation (Lewin et al. 2012) [Model]

Lombardi A, Jedlicka P, Luhmann HJ, Kilb W (2019) Interactions Between Membrane Resistance, GABA-A Receptor Properties, Bicarbonate Dynamics and Cl-Transport Shape Activity-Dependent Changes of Intracellular Cl- Concentration Int J of Mol Sci [Journal] [PubMed]

•	Factors contribution to GDP-induced [Cl-]i transients (Lombardi et al 2019) [Model]

Prescott SA, Sejnowski TJ, De Koninck Y (2006) Reduction of anion reversal potential subverts the inhibitory control of firing rate in spinal lamina I neurons: towards a biophysical basis for neuropathic pain. Mol Pain 2:32 [Journal] [PubMed]

Ratté S, Prescott SA (2011) ClC-2 channels regulate neuronal excitability, not intracellular chloride levels. J Neurosci 31:15838-43 [Journal] [PubMed]

•	ClC-2 channels regulate neuronal excitability, not intracellular Cl- levels (Ratte & Prescott 2011) [Model]

Sudhakar SK, Choi TJ, Ahmed OJ (2019) Biophysical Modeling Suggests Optimal Drug Combinations for Improving the Efficacy of GABA Agonists after Traumatic Brain Injuries. J Neurotrauma 36:1632-1645 [Journal] [PubMed]

•	Biophysical modeling of pathological brain states (Sudhakar et al 2019) [Model]