Models that contain the Model Type : Neuromuscular Junction

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    Models   Description
1. Facilitation by residual calcium (Stockbridge, Hines 1982)
The residual calcium hypothesis is compatible with facilitation of transmitter release from the neuromuscular junction.
2. Model of peristalsis in the dorsal blood vessel of Lumbriculus variegatus (Halfmann and Crisp 2011)
The blackworm, Lumbriculus variegatus, has a segmented dorsal blood vessel that acts as a peristaltic pump to move blood through its closed circulatory system. Here, we conducted a kinematic study using videography and computational modeling as a first step toward understanding the control of DBV pulsation. A simple feed-forward system of distributed, coupled neuronal oscillators is a sufficient model was a sufficient model to explain the control of pulsation in the blackworm.
3. Motoneuron simulations for counting motor units (Major and Jones 2005)
Simulations of clinical methods to count the number of motoneurons/motor units in human patients. Models include stimulation of motor axons or voluntary activation and responses are measured as muscle tension or EMG.
4. Nerve terminal currents at lizard neuromuscular junction (Lindgren, Moore 1989)
Loose patch clamp measurement of presynaptic ionic currents at lizard neuromuscular junction compared with computer simulations.
5. Presynaptic calcium dynamics at neuromuscular junction (Stockbridge, Moore 1984)
The diffusion of calcium is effectively reduced by the ratio of bound to free calcium. Treating the release magnitude as proportional to the fourth power of calcium concentration next to the membrane gives reasonable facilitation with very little release between spikes.
6. Quantal neurotransmitter release kinetics with fixed and mobile Ca2+ buffers (Gilmanov et al. 2008)
"... In this work, we used computer modeling of quanta release kinetics with various levels of calcium influx and in the presence of endogenous calcium buffers with varying mobilities. The results of this modeling revealed the desynchronization of quanta release under low calcium influx in the presence of an endogenous fixed calcium buffer, with a diffusion coefficient much smaller than that of free Ca2+, and synchronization occurred upon adding a mobile buffer. This corresponds to changes in secretion time course parameters found experimentally ..."

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