| || Models ||Description|
AP initiation and propagation in type II cochlear ganglion cell (Hossain et al 2005)
||The model of type II cochlear ganglion cell was based on the
immunostaining of the mouse auditory pathway. Specific antibodies were
used to map the distribution of voltage-dependent sodium channels along
the two unmyelinated axon-like processes of the bipolar ganglion cells.
Three distinct hot spots were detected. A high density of sodium
channels was present over the entire trajectory of sensory endings
beneath the outer hair cells (the most distal portion of the peripheral
axon). The other two hot spots were localized in the initial segments of
both of the axons that flank the unmyelinated bipolar ganglion cell bodies.
A biophysical model indicates that all three hot spots might play
important roles in action potential initiation and propagation. For
instance, the hot spot in the receptor segment is important for
transforming the receptor potentials into a full blown action potential
(Supplemental Fig. 1). The hot spots in the two paraganglionic axon
initial segments are there to ensure the successful propagation of
action potentials from the peripheral to the central axon through the
The Readme.txt file provides step by step instructions on how to
recreate Figures 6 and 7 of Hossain et al., 2005 paper.
Oscillating neurons in the cochlear nucleus (Bahmer Langner 2006a, b, and 2007)
||"Based on the physiological and anatomical data, we propose a model consisting of a minimum network of two choppers that are interconnected with a synaptic delay of 0.4 ms (Bahmer and Langner 2006a) . Such minimum delays have been found in different systems and in various animals (e.g. Hackett, Jackson, and Rubel 1982; Borst, Helmchen, and Sakmann 1995). The choppers receive input from both the auditory nerve and an onset neuron. This model can reproduce the mean, standard deviation, and coefficient of variation of the ISI and the dynamic features of AM coding of choppers."