Citation Relationships



Crunelli V, Leresche N (2002) Childhood absence epilepsy: genes, channels, neurons and networks. Nat Rev Neurosci 3:371-82 [PubMed]

References and models cited by this paper

References and models that cite this paper

Broicher T, Kanyshkova T, Landgraf P, Rankovic V, Meuth P, Meuth SG, Pape HC, Budde T (2007) Specific expression of low-voltage-activated calcium channel isoforms and splice variants in thalamic local circuit interneurons. Mol Cell Neurosci 36:132-45 [Journal] [PubMed]

Broicher T, Kanyshkova T, Meuth P, Pape HC, Budde T (2008) Correlation of T-channel coding gene expression, IT, and the low threshold Ca2+ spike in the thalamus of a rat model of absence epilepsy. Mol Cell Neurosci 39:384-99 [Journal] [PubMed]

Broicher T, Seidenbecher T, Meuth P, Munsch T, Meuth SG, Kanyshkova T, Pape HC, Budde T (2007) T-current related effects of antiepileptic drugs and a Ca2+ channel antagonist on thalamic relay and local circuit interneurons in a rat model of absence epilepsy. Neuropharmacology 53:431-46 [Journal] [PubMed]

Chen M, Guo D, Li M, Ma T, Wu S, Ma J, Cui Y, Xia Y, Xu P, Yao D (2015) Critical Roles of the Direct GABAergic Pallido-cortical Pathway in Controlling Absence Seizures. PLoS Comput Biol 11:e1004539 [Journal] [PubMed]

   Basal ganglia-corticothalamic (BGCT) network (Chen et al., 2014) [Model]

Chen M, Guo D, Wang T, Jing W, Xia Y, Xu P, Luo C, Valdes-Sosa PA, Yao D (2014) Bidirectional control of absence seizures by the basal ganglia: a computational evidence. PLoS Comput Biol 10:e1003495 [Journal] [PubMed]

   Basal ganglia-corticothalamic (BGCT) network (Chen et al., 2014) [Model]

Hall S, Hunt M, Simon A, Cunnington LG, Carracedo LM, Schofield IS, Forsyth R, Traub RD, Whittington MA (2015) Unbalanced Peptidergic Inhibition in Superficial Neocortex Underlies Spike and Wave Seizure Activity. J Neurosci 35:9302-14 [Journal] [PubMed]

   Unbalanced peptidergic inhibition in superficial cortex underlies seizure activity (Hall et al 2015) [Model]
   Electrodecrements in in vitro model of infantile spasms (Traub et al 2020) [Model]

Hall SP, Traub RD, Adams NE, Cunningham MO, Schofield I, Jenkins AJ, Whittington MA (2018) Enhanced interlaminar excitation or reduced superficial layer inhibition in neocortex generates different spike-and-wave-like electrographic events in vitro. J Neurophysiol 119:49-61 [Journal] [PubMed]

   The origin of different spike and wave-like events (Hall et al 2017) [Model]

Hayut I, Fanselow EE, Connors BW, Golomb D (2011) LTS and FS inhibitory interneurons, short-term synaptic plasticity, and cortical circuit dynamics. PLoS Comput Biol 7:e1002248 [Journal] [PubMed]

   Rate model of a cortical RS-FS-LTS network (Hayut et al. 2011) [Model]

Liu Y, Milton J, Campbell SA (2019) Outgrowing seizures in Childhood Absence Epilepsy: time delays and bistability. J Comput Neurosci [Journal] [PubMed]

   Thalamocortical loop with delay for investigation of absence epilepsy (Liu et al 2019) [Model]

Suffczynski P, Kalitzin S, Lopes Da Silva FH (2004) Dynamics of non-convulsive epileptic phenomena modeled by a bistable neuronal network. Neuroscience 126:467-84 [Journal] [PubMed]

   Thalamocortical model of spike and wave seizures (Suffczynski et al. 2004) [Model]

Vitko I, Chen Y, Arias JM, Shen Y, Wu XR, Perez-Reyes E (2005) Functional characterization and neuronal modeling of the effects of childhood absence epilepsy variants of CACNA1H, a T-type calcium channel. J Neurosci 25:4844-55 [Journal] [PubMed]

   T channel currents (Vitko et al 2005) [Model]

Zhao X, Robinson PA (2015) Generalized seizures in a neural field model with bursting dynamics. J Comput Neurosci 39:197-216 [Journal] [PubMed]

   Neural-field model of generalized seizures (Zhao et al., 2015) [Model]

(12 refs)