231. Vigot R and Batini C. (1997) GABA(B) receptor activation of Purkinje cells in cerebellar slices. Neurosci Res 29:151-60.

NeuronCompartmentPropertyConnectivityNotes
Cerebellum purkinje cellDistal equivalent dendriteGabaAStellate Cell terminals (T)Recordings in slices showed that GABA inhibition was mediated by GABA(B) receptors in the dendrites and GABA(A) receptors in the soma and dendrites (Vigot R and Batini C, 1997231 ).
Cerebellum purkinje cellMiddle equivalent dendriteGabaAStellate Cell terminals (T)Recordings in slices showed that GABA inhibition was mediated by GABA(B) receptors in the dendrites and GABA(A) receptors in the soma and dendrites (Vigot R and Batini C, 1997231 ).
Cerebellum purkinje cellProximal equivalent dendriteGabaAStellate Cell terminals (T)Recordings in slices showed that GABA inhibition was mediated by GABA(B) receptors in the dendrites and GABA(A) receptors in the soma and dendrites (Vigot R and Batini C, 1997231 ).
Cerebellum purkinje cellDistal equivalent dendriteGabaB.>232< found evidence for the presence of GABAB receptors on cell dendrites. Recordings in slices showed that GABA inhibition was mediated by GABA(B) receptors in the dendrites and GABA(A) receptors in the soma and dendrites. Therefore, the GABA released by stellate cells modulates Purkinje cells activity through two inhibitory mechanisms (>231<). The cellular localization of GABAB binding was investigated using lesion techniques. It was suggested that the majority of cerebellar molecular layer GABAB binding sites are located on Purkinje cell dendrites. During development binding in the molecular layer peaks between postnatal day 14 and postnatal day 28 and then decreases to adult levels (>235<).
Cerebellum purkinje cellMiddle equivalent dendriteGabaB.>232< found evidence for the presence of GABAB receptors on cell dendrites. Recordings in slices showed that GABA inhibition was mediated by GABA(B) receptors in the dendrites and GABA(A) receptors in the soma and dendrites. Therefore, the GABA released by stellate cells modulates Purkinje cells activity through two inhibitory mechanisms (>231<). The cellular localization of GABAB binding was investigated using lesion techniques. It was suggested that the majority of cerebellar molecular layer GABAB binding sites are located on Purkinje cell dendrites. During development binding in the molecular layer peaks between postnatal day 14 and postnatal day 28 and then decreases to adult levels (>235<).
Cerebellum purkinje cellProximal equivalent dendriteGabaB.>232< found evidence for the presence of GABAB receptors on cell dendrites. Recordings in slices showed that GABA inhibition was mediated by GABA(B) receptors in the dendrites and GABA(A) receptors in the soma and dendrites. Therefore, the GABA released by stellate cells modulates Purkinje cells activity through two inhibitory mechanisms (>231<). The cellular localization of GABAB binding was investigated using lesion techniques. It was suggested that the majority of cerebellar molecular layer GABAB binding sites are located on Purkinje cell dendrites. During development binding in the molecular layer peaks between postnatal day 14 and postnatal day 28 and then decreases to adult levels (>235<).
Cerebellum purkinje cellSomaGabaA.Recordings in slices showed that GABA inhibition was mediated by GABA(B) receptors in the dendrites and GABA(A) receptors in the soma and dendrites (Vigot R and Batini C, 1997231 ).

References
231. Vigot R and Batini C. (1997) GABA(B) receptor activation of Purkinje cells in cerebellar slices. Neurosci Res 29:151-60.
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