1. Turgeon SM and Albin RL. (1993) Pharmacology, distribution, cellular localization, and development of GABAB binding in rodent cerebellum. Neuroscience 55:311-23.

NeuronCompartmentPropertyConnectivityNotes
Cerebellum Purkinje GABA cellDistal equivalent dendriteGabaB.found evidence for the presence of GABAB receptors on cell dendrites. Recordings in slices showed that GABA inhibition was mediated by GABA(B) receptors in the dendrites and GABA(A) receptors in the soma and dendrites. Therefore, the GABA released by stellate cells modulates Purkinje cells activity through two inhibitory mechanisms (Vigot R and Batini C, 19972 ). The cellular localization of GABAB binding was investigated using lesion techniques. It was suggested that the majority of cerebellar molecular layer GABAB binding sites are located on Purkinje cell dendrites. During development binding in the molecular layer peaks between postnatal day 14 and postnatal day 28 and then decreases to adult levels (Turgeon SM and Albin RL, 19931 ).
Cerebellum Purkinje GABA cellMiddle equivalent dendriteGabaB.found evidence for the presence of GABAB receptors on cell dendrites. Recordings in slices showed that GABA inhibition was mediated by GABA(B) receptors in the dendrites and GABA(A) receptors in the soma and dendrites. Therefore, the GABA released by stellate cells modulates Purkinje cells activity through two inhibitory mechanisms (Vigot R and Batini C, 19972 ). The cellular localization of GABAB binding was investigated using lesion techniques. It was suggested that the majority of cerebellar molecular layer GABAB binding sites are located on Purkinje cell dendrites. During development binding in the molecular layer peaks between postnatal day 14 and postnatal day 28 and then decreases to adult levels (Turgeon SM and Albin RL, 19931 ).
Cerebellum Purkinje GABA cellProximal equivalent dendriteGabaB.found evidence for the presence of GABAB receptors on cell dendrites. Recordings in slices showed that GABA inhibition was mediated by GABA(B) receptors in the dendrites and GABA(A) receptors in the soma and dendrites. Therefore, the GABA released by stellate cells modulates Purkinje cells activity through two inhibitory mechanisms (Vigot R and Batini C, 19972 ). The cellular localization of GABAB binding was investigated using lesion techniques. It was suggested that the majority of cerebellar molecular layer GABAB binding sites are located on Purkinje cell dendrites. During development binding in the molecular layer peaks between postnatal day 14 and postnatal day 28 and then decreases to adult levels (Turgeon SM and Albin RL, 19931 ).

Classical References: first publications on each compartmental property; search PubMed for complete list
1.  Turgeon SM and Albin RL. (1993) Pharmacology, distribution, cellular localization, and development of GABAB binding in rodent cerebellum. Neuroscience 55:311-23.
2.  Vigot R and Batini C. (1997) GABA(B) receptor activation of Purkinje cells in cerebellar slices. Neurosci Res 29:151-60.