Models that contain the Model Concept : Aging/Alzheimer`s

(Alzheimer's is a progressive and terminal incurable disease. Most prevalent in people over 65 years old with increasing incidence with age. The first symptom is short-term memory loss. Over subsequent years confusion, anger, mood swings, general cognitive decline and memory loss are the usual course. The course of the disease is typically 5 to 20 years.) Re-display model names with descriptions

	Models
1.	A model of ventral Hippocampal CA1 pyramidal neurons of Tg2576 AD mice (Spoleti et al. 2021)
2.	A Neural mass computational model of the Thalamocorticothalamic circuitry (Bhattacharya et al. 2011)
3.	Age-dependent excitability of CA1 pyramidal neurons in APPPS1 Alzheimer's model (Vitale et al 2021)
4.	Amyloid beta (IA block) effects on a model CA1 pyramidal cell (Morse et al. 2010)
5.	Amyloid-beta effects on release probability and integration at CA3-CA1 synapses (Romani et al. 2013)
6.	Axonal spheroids and conduction defects in Alzheimer’s disease (Yuan, Zhang, Tong, et al 2022)
7.	Biochemical Systems Theory Model of TNFa related pathways (Sasidharakurup and Diwakar 2020)
8.	CA1 pyramidal neurons: effects of Alzheimer (Culmone and Migliore 2012)
9.	Computational neuropharmacology of CA1 pyramidal neuron (Ferrante et al. 2008)
10.	Effects of increasing CREB on storage and recall processes in a CA1 network (Bianchi et al. 2014)
11.	Electrostimulation to reduce synaptic scaling driven progression of Alzheimers (Rowan et al. 2014)
12.	Human L5 Cortical Circuit (Guet-McCreight)
13.	Modelling reduced excitability in aged CA1 neurons as a Ca-dependent process (Markaki et al. 2005)
14.	Rhesus Monkey Layer 3 Pyramidal Neurons: Young vs aged PFC (Coskren et al. 2015)
15.	Rhesus Monkey Young and Aged L3 PFC Pyramidal Neurons (Rumbell et al. 2016)
16.	The APP in C-terminal domain alters CA1 neuron firing (Pousinha et al 2019)
17.	Tonic activation of extrasynaptic NMDA-R promotes bistability (Gall & Dupont 2020)

Re-display model names with descriptions