ModelDB: Spine neck plasticity controls postsynaptic calcium signals (Grunditz et al. 2008)

Spine neck plasticity controls postsynaptic calcium signals (Grunditz et al. 2008)

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Accession:116769

This model was set up to dissect the relative contribution of different channels to the spine calcium transients measured at single spines.
Reference:
1 . Grunditz A, Holbro N, Tian L, Zuo Y, Oertner TG (2008) Spine neck plasticity controls postsynaptic calcium signals through electrical compartmentalization. J Neurosci 28:13457-66 [PubMed]

Model Information (Click on a link to find other models with that property)

Model Type:	Synapse;
Brain Region(s)/Organism:
Cell Type(s):	Hippocampus CA1 pyramidal GLU cell;
Channel(s):	I Calcium; I R;
Gap Junctions:
Receptor(s):	AMPA; NMDA;
Gene(s):
Transmitter(s):	Glutamate;
Simulation Environment:	NEURON;
Model Concept(s):	Calcium dynamics;
Implementer(s):	Mueller, Asa [asa.mueller at fmi.ch];